Javier Egea is Senior Researcher at Instituto Investigación Sanitaria del Hospital Universitario de la Princesa (Madrid, Spain). The core research line is based on the study of the mechanisms that trigger neuroinflammation at the CNS level, in their control to identify new targets to offer neuroprotection, and in the search for prognostic biomarkers related to inflammation and oxidative stress, with emphasis on pathologies such as stroke and traumatic brain injury, and the study of how melatonin could change the progression of this diseases. Specifically, our group focuses on the study of the mechanisms that control the activation of the innate immune system (NLRP3 inflammasome). Dr. Egea has been involved in 14 research projects, he has written 3 book chapters and he has published seventy papers in JCR international peer-reviewed journals. He currently belongs to the editorial board in the journal “Oxidative Medicine and Cellular Longevity” (ISSN: 1942-0900).

Javier Egea, PhD Senior Researcher National Health System “Miguel Servet II,” Instituto de Investigación Sanitaria del H.U. La Princesa, Spain

Javier Egea is Senior Researcher at Instituto Investigación Sanitaria del Hospital Universitario de la Princesa (Madrid, Spain). The core research line is based on the study of the mechanisms that trigger neuroinflammation at the CNS level, in their control to identify new targets to offer neuroprotection, and in the search for prognostic biomarkers related to inflammation and oxidative stress, with emphasis on pathologies such as stroke and traumatic brain injury, and the study of how melatonin could change the progression of this diseases. Specifically, our group focuses on the study of the mechanisms that control the activation of the innate immune system (NLRP3 inflammasome). Dr. Egea has been involved in 14 research projects, he has written 3 book chapters and he has published seventy papers in JCR international peer-reviewed journals. He currently belongs to the editorial board in the journal “Oxidative Medicine and Cellular Longevity” (ISSN: 1942-0900).

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