Effects of Iodine and Selenium on the Thyroid Function

Inés Velasco, Teresa Arrobas, Elena Marina, Federico Soriguer and Jose Luis Gómez-Ariza
Servicio de Ginecología y Obstetricia. Hospital de Riotinto, Huelva, Spain

Series: Endocrinology Research and Clinical Developments
BISAC: MED027000

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Volume 10

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Special issue: Resilience in breaking the cycle of children’s environmental health disparities
Edited by I Leslie Rubin, Robert J Geller, Abby Mutic, Benjamin A Gitterman, Nathan Mutic, Wayne Garfinkel, Claire D Coles, Kurt Martinuzzi, and Joav Merrick

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Iodine and selenium are two trace elements essentials to human life due to their implication in normal thyroid function. Iodine is required by thyroid gland to synthesize thyroxine (T4, tetraiodothyroxine) and triiodothyronine (T3). Iodine deficiency is still a worldwide Public Health problem, with over 1,200 millions of habitans exposed to risk because that daily iodine intake does not reach optimal levels. Although both isolated deficiency of iodine and selenium can cause diseases themselves, combined deficiency of these micronutrients promotes the cell damage, fibrotic degeneration and complete atrophy of thyroid gland typical of the myxedematous cretinism, where iodine supplementation alone cannot restore thyroid function. Clinical implications of deficiency and supplementation of these two micronutrients should be clarified in order to develop preventive strategies and dietary recommendations to population. (Imprint: Nova)

Abstract

Iodine

Natural Sources of Iodine

Iodine Metabolism

Minimum Daily Iodine Intake Recommendations

Iodine Deficiency Disorders (IDD)

Scope of the Problem

Severity of the Endemia

Indicators Defining Iodine Deficit

Special At-Risk Population

Selenium (Se)

Natural Sources of Selenium

Dietary Selenium Requirements

Metabolism of Selenium

Selenoproteins

Iodine, Selenium and the Thyroid Function

References

Index

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