Table of Contents
Preface
Chapter 1. Active Participation of Dendritic Cells in Tumor Cell Immune Evasion
Chapter 2. Aging-Induced Aberrant Dendritic Cells in Terms of Highly Proliferative Tumor Cell Clonal Subsets
Chapter 3. Sustained Metabolic System Imbalance as Induced Immune Suppression of Dendritic Cells in Tumor Neoantigenicity Profiles
Chapter 4. Inclusive Loss of Integrity of The Antitumor Immune Response as Chemokine Patterns of Nonrecognition
Chapter 5. Incongruent Biology of Redistributed Chemokine Action in Carcinogenesis
Chapter 6. The Nature of Modulated Redistribution of Integral Immunity as System Redefinition of Clinical Non-Responsiveness to the Antitumor Antigenicity
Chapter 7. Complexity of Inter-Activities of Suppressed Immune Response with Dendritic Cell Development and Differentiation in Tumors
Chapter 8. System Profiles of Dendritic Cell Motility as Modeled Tumor Cell Proliferation and Spread
Chapter 9. Duality of Unstable Dynamics of Emerging Tumor Cells and of Infiltrating Dendritic Cells in Carcinogenesis
Chapter 10. Systems of Predetermination of Cytokine/Chemokine Action Towards Cancer Antigenicity in Terms of Dendritic Cell Plasticity
Chapter 11. Exosome System Preferences as Integral to the Immune Response and Immune Non-Response to Tumor Neoantigens
Chapter 12. Constitution of a Failed Antitumor Immune Response as Carcinogenesis Sustainment
Chapter 13. Multiple Models of Failed Immune Responses Redefine Carcinogenesis
Chapter 14. Effect Dominance of Integral Cytokine/Chemokine Networks in Tumor Cell Antigenicity Response
Chapter 15. Incongruent Dislocation of Epitope Spread as Immunosuppression Dynamics of Tumor Cell Clones
Chapter 16. Systems of Immune Evasion are Inherent to the Inflammatory Response to Tumorigenesis
Chapter 17. Heterogeneous Dendritic Cell Subset Interchange as Tumor Immunosuppression
Chapter 18. Recurrent Immune Responses Are Primary Reactivity to the Tumor Microenvironment Rather Than to the Integral Tumor Cells Themselves
Chapter 19. The Immunologic Non-Response is a Physiologic, Conditional and Homeostatic State of an Integral Immune System Towards Tumor Antigenicity
Chapter 20. Dendritic Cell Plasticity as History of Performance Dynamics
Chapter 21. Contact Mechanics of Immune Evasion Involve Suppression of The Antigen-Presentation Step and of the Tumor-Mirrored Migration of the Dendritic Cells
Chapter 22. Microenvironmental Reproducible Immunomodulation as Proposed Growth and Spread of Malignant Cells in Terms of Neoantigenicity
Chapter 23. Tumor Cell Necrosis Modulates the Immune Tolerance Induced by the Tumor Microenvironment
Index